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Verfasst von:Paine, Nicola J. [VerfasserIn]   i
 Ring, Christopher [VerfasserIn]   i
 Aldred, Sarah [VerfasserIn]   i
 Bosch, Jos A. [VerfasserIn]   i
 Wadley, Alex J. [VerfasserIn]   i
 Veldhuijzen van Zanten, Jet J. C. S. [VerfasserIn] [VerfasserIn]   i
Titel:Eccentric-exercise induced inflammation attenuates the vascular responses to mental stress
Verf.angabe:Nicola J. Paine, Christopher Ring, Sarah Aldred, Jos A. Bosch, Alex J. Wadley, Jet J.C.S. Veldhuijzen van Zanten
E-Jahr:2013
Jahr:30 January 2013
Umfang:10 S.
Fussnoten:Gesehen am 31.03.2022
Titel Quelle:Enthalten in: Brain, behavior and immunity
Ort Quelle:Orlando, Fla. [u.a.] : Elsevier, 1987
Jahr Quelle:2013
Band/Heft Quelle:30(2013), Seite 133-142
ISSN Quelle:1090-2139
Abstract:Mental stress has been identified as a trigger of myocardial infarction (MI), with inflammation and vascular responses to mental stress independently implicated as contributing factors. This study examined whether inflammation moderates the vascular responses to mental stress. Eighteen healthy male participants completed a stress task under two counter balanced conditions. In the exercise condition, a morning bout of eccentric exercise (12×5 repetitions of unilateral eccentric knee extension at 120% intensity of concentric one repetition maximum) was used to increase levels of inflammatory-responsive cytokines during an afternoon stress session scheduled 6h later. In the control condition, participants sat and relaxed for 45min, 6h prior to the afternoon stress session. Forearm blood flow, calf blood flow (measured in the leg which completed the exercise task), blood pressure, heart rate and cardiac output were assessed at rest and in response to mental stress. As expected, interleukin-6 was higher (p=.02) 6h post exercise, i.e., at the start of the stress session, as compared to the no-exercise control condition. Mental stress increased forearm blood flow, calf blood flow, blood pressure, heart rate, and cardiac output in both conditions (p’s<.001). Stress-induced calf blood flow was attenuated in the exercise condition compared to the control condition (p<.05) which was not the case for forearm blood flow. This study found that the inflammatory response to eccentric exercise attenuated the vascular responses to mental stress locally at the site of eccentric exercise-induced inflammation. The observed impairment in vascular responses to stress associated with increased levels of inflammation suggests a mechanism through which inflammation might increase the risk for MI.
DOI:doi:10.1016/j.bbi.2013.01.082
URL:Bitte beachten Sie: Dies ist ein Bibliographieeintrag. Ein Volltextzugriff für Mitglieder der Universität besteht hier nur, falls für die entsprechende Zeitschrift/den entsprechenden Sammelband ein Abonnement besteht oder es sich um einen OpenAccess-Titel handelt.

Volltext: https://doi.org/10.1016/j.bbi.2013.01.082
 Volltext: https://www.sciencedirect.com/science/article/pii/S0889159113000858
 DOI: https://doi.org/10.1016/j.bbi.2013.01.082
Datenträger:Online-Ressource
Sprache:eng
Sach-SW:Eccentric exercise
 Inflammation
 Mental stress
 Stress-induced vasodilation
 Vascular responses
K10plus-PPN:1797151339
Verknüpfungen:→ Zeitschrift

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