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Status: Bibliographieeintrag

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Verfasst von:Hamann, Ute [VerfasserIn]   i
 Herbold, Christian [VerfasserIn]   i
 Costa, Serban-Dan [VerfasserIn]   i
 Solomayer, Erich-Franz [VerfasserIn]   i
 Kaufmann, Manfred [VerfasserIn]   i
 Bastert, Gunther [VerfasserIn]   i
 Ulmer, Hans Ulrich [VerfasserIn]   i
 Frenzel, Hans [VerfasserIn]   i
 Komitowski, Dymitr [VerfasserIn]   i
Titel:Allelic imbalance on chromosome 13q
Titelzusatz:evidence for the involvement of BRCA2 and RB1 in sporadic breast cancer
Verf.angabe:Ute Hamann, Christian Herbold, Serban Costa, Erich-Franz Solomayer, Manfred Kaufmann, Gunther Bastert, Hans Ulrich Ulmer, Hans Frenzel, Dymitr Komitowski
E-Jahr:1996
Jahr: May 01 1996
Umfang:3 S.
Fussnoten:Gesehen am 27.02.2024
Titel Quelle:Enthalten in: Cancer research
Ort Quelle:Philadelphia, Pa. : AACR, 1916
Band/Heft Quelle:56(1996), 9, Seite 1988-1990
ISSN Quelle:1538-7445
Abstract:Recently, the breast cancer susceptibility gene BRCA2 has been identified in chromosome 13q, a region that also contains the retinoblastoma gene RB1. To elucidate a possible role of BRCA2 and RB1 in sporadic breast tumorigenesis, allelic imbalance (AI) at 13q loci was examined in 78 primary sporadic breast tumors. AI was found in 52-63% of tumors. Nine tumors showed AI only in the BRCA2 region but not at RB1. Six tumors showed AI at RB1 but not in the BRCA2 region. AI in the BRCA2 region correlated significantly with aneuploidy (P = 0.032) and AI at RB1 with small tumor size (P = 0.025). Our data suggest that BRCA2 and RB1 may be both distinct target loci for AI on chromosome 13 in sporadic breast cancer.
Datenträger:Online-Ressource
Sprache:eng
K10plus-PPN:188171179X
Verknüpfungen:→ Zeitschrift

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