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Verfasst von:Itzel, Timo [VerfasserIn]   i
 Falconer, Thomas [VerfasserIn]   i
 Daza Barragán, Jimmy Andres [VerfasserIn]   i
 Roig, Ana [VerfasserIn]   i
 Park, Jimyung [VerfasserIn]   i
 Cheong, Jae Youn [VerfasserIn]   i
 Park, Rae Woong [VerfasserIn]   i
 Wiest, Isabella [VerfasserIn]   i
 Ebert, Matthias [VerfasserIn]   i
 Hripcsak, George [VerfasserIn]   i
 Teufel, Andreas [VerfasserIn]   i
Titel:Letter to the editor: vaccination against upper respiratory infections is a matter of survival in alcoholic liver disease$ppostscript : letters
Verf.angabe:Timo Itzel, Thomas Falconer, Jimmy Daza, Ana Roig, Jimyung Park, Jae Youn Cheong, Rae Woong Park, Isabella Wiest, Matthias Ebert, George Hripcsak, Andreas Teufel
E-Jahr:2023
Jahr:January 2023
Umfang:2 S.
Fussnoten:Gesehen am 01.07.2024
Titel Quelle:Enthalten in: Gut
Ort Quelle:London : BMJ Publishing Group, 1960
Jahr Quelle:2023
Band/Heft Quelle:72(2023), 1 vom: Jan., Seite 208-209
ISSN Quelle:1468-3288
Abstract:We read with great interest the report on SARS-CoV-2 infection in patients with primary biliary cholangitis (PBC) by Ampuero et al .1 An independent association of chronic liver disease with adverse clinical outcome in patients with COVID-19 was previously reported in this journal.2 However, the impact of SARS-CoV-2 and upper airway infections in general on patients with alcoholic liver disease (ALD) remains widely unknown. Yet, those data would be of high relevance to global public health and vaccination campaigns since alcohol related diseases are endemic. 57% of the population over age 15 has consumed alcohol in the last 12 months, and 2.3 billion people are current drinkers.3 - - With respect to upper airway respiratory infections, patients with chronic liver disease, including those with ALD, are particularly susceptible to infections as the immune system is dysfunctional through several pathological mechanisms including decreased opsonisation, reticuloendothelial dysfunction, neutrophils impairment and abnormal immunoglobulin synthesis.4 5 Moreover, alcohol abuse, independently from other factors, depresses the immune system by decreasing the lymphocytes and antibodies and the production of cytokines as TNF-alpha, IL-1 and IL-6.6 Several human and murine studies …
DOI:doi:10.1136/gutjnl-2022-327086
URL:Bitte beachten Sie: Dies ist ein Bibliographieeintrag. Ein Volltextzugriff für Mitglieder der Universität besteht hier nur, falls für die entsprechende Zeitschrift/den entsprechenden Sammelband ein Abonnement besteht oder es sich um einen OpenAccess-Titel handelt.

kostenfrei: Volltext: https://doi.org/10.1136/gutjnl-2022-327086
 kostenfrei: Volltext: http://gut.bmj.com/content/72/1/208
 DOI: https://doi.org/10.1136/gutjnl-2022-327086
Datenträger:Online-Ressource
Sprache:eng
Sach-SW:ALCOHOLIC LIVER DISEASE
K10plus-PPN:1892775786
Verknüpfungen:→ Zeitschrift

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